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. 2020 Nov 23;71(5):1061–1069. doi: 10.1007/s12031-020-01728-z

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Association between rs3826795 polymorphism and levels of Hcy and NEFA. a An increased level of Hcy in IS patients compared to controls (P < 0.001). b Increased level of NEFA in IS patients compared to controls (P < 0.001). c Increased level of Hcy in IS patients carrying the rs3826795 AA compared to those carrying the rs3826795 AG+GG (P = 0.024). d–f The levels of Hcy and NEFA showed no significant differences among different genotype groups (P > 0.05). IS ischemic stroke, Hcy homocysteic acid, NEFA non-esterified fatty acid