Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2021 Apr 2;10(4):555. doi: 10.3390/antiox10040555

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© 2021 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

PMC Copyright notice

Impact of GSNOR inhibitor on cardiac damage in ischemia–reperfusion. (A), levels of creatine kinase release in the coronary effluent of Langendorff perfused hearts in the presence of increasing concentrations of the GSNOR inhibitor C2 and controls (vehicle). (B), representative images of hearts stained with TTC after being submitted to a protocol of ischemia–reperfusion, under control conditions or in the presence of the GSNOR inhibitor C2 at 0.1, 1 and 10 µmol/L. Right, graph depicting the analysis of the infarcted area. Asterisk (*) indicates p < 0.05 and **, p < 0.005 compared to vehicle (control), by two-way ANOVA (creatine kinase) and one-way ANOVA with multiple comparisons for infarct size analysis. n = 5 in each group.