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. 2021 Apr 28;27(16):1785–1804. doi: 10.3748/wjg.v27.i16.1785

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©The Author(s) 2021. Published by Baishideng Publishing Group Inc. All rights reserved.

This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial.

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Knockdown of MRPL35 inhibits gastric carcinoma cell proliferation and monoclonal formation and promotes gastric carcinoma cell apoptosis. A and B: The Celigo cell count assay was used to analyze the proliferation of AGS cells (A) and HGC-27 cells (B) transfected with shCtrl (negative control virus) and shMRPL35 (lentiviral particles of MRPL35); C and D: Colony formation assay of shCtrl and shMRPL35 infected AGS cells (C) and HGC-27 cells (D); E and F: Flow cytometry analysis of apoptosis of shCtrl and shMRPL35 infected AGS cells (E) and HGC-27 cells (F). n = 3. ^bP < 0.01. shCtrl: Negative control virus; shMRPL35: Lentiviral particles of MRPL35.