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. 2021 Apr 16;26(8):2313. doi: 10.3390/molecules26082313

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© 2021 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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(A,B) The histological analysis of BRAs on subacute (A) and acute (B) alcohol-induced liver injury (×500 magnification): (a) control group, (b) model group, (c) low-dose BRA group, (d) middle-dose BRA group, and (e) high-dose BRA group. (C) NF-κB and TGF-β expression in the liver tissue. (D) Morphological changes in HepG2 cells. (E) The induction of apoptosis was determined by flow cytometric analysis with Annexin V-FITC and PI-staining. (F) Effects of cyanidin-3-O-rutinoside and BRAs on the expression of various cell apoptosis proteins in HepG2 cells.

(A,B) The histological analysis of BRAs on subacute (A) and acute (B) alcohol-induced liver injury (×500 magnification): (a) control group, (b) model group, (c) low-dose BRA group, (d) middle-dose BRA group, and (e) high-dose BRA group. (C) NF-κB and TGF-β expression in the liver tissue. (D) Morphological changes in HepG2 cells. (E) The induction of apoptosis was determined by flow cytometric analysis with Annexin V-FITC and PI-staining. (F) Effects of cyanidin-3-O-rutinoside and BRAs on the expression of various cell apoptosis proteins in HepG2 cells.