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. 2021 Apr 26;21:460. doi: 10.1186/s12885-021-08210-y

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Construction of prognostic-related transcriptional dysregulation network of KD genes. AB, Examples show the prognostic efficacy of KD genes ABCE1 (a) and SHFM1 (b) gastric cancer patients. c, Genomic variation of all recognized KD genes in gastric cancer patients, including high copy number copy (dark red), low level amplification (bright red), homozygous copy number deletion (dark blue), heterozygous deletion (Bright blue), as well as somatic mutation (green). d, The transcriptional dysregulation network of KD genes. e, The number of dysregulation factors for KD genes