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. 2021 Apr 26;22:121. doi: 10.1186/s12931-021-01728-6

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — The long-term effect of postnatal hypoxia on the pulmonary vasculature of adult rats. a A comparison of lung/body wet weight ratio in adult rats who had been exposed to postnatal hypoxia (N = 6 per group). b Right ventricular hypertrophy (Fulton) index [RV / (LV + S)] differed between hypoxia and normoxia rats at 9 weeks old (N = 6–7 per group). c Comparison of mean pulmonary arterial pressure (mPAP) between the groups (N = 5–6 per group). d Immunohistochemical staining of lung tissue and the thickness of pulmonary arterial intima measured by α-SMA (N = 5 per group). e Western blot images of PH-related proteins in the two groups (N = 6 per group). f–i Quantitative analysis of the Western blot experiments (N = 6 per group). *P < 0.05, ** P < 0.01 compared to the control group