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Post-ischemic BDNF-TrkB signaling is proposed to facilitate the emergence of phenobarbital unresponsive neonatal seizures. (A) Schematized inhibitory synapses in a non-pathological neonatal brain, (B) post-ischemic insult, and (C) ANA12 treated post-ischemic insult. (D) Legend for proteins, molecules, and cell types of interest. An improved understanding of the multiples levels of BDNF regulation in neonatal seizures is necessary.
