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. 2020 Oct 15;52(10):1754–1765. doi: 10.1038/s12276-020-00514-6

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a Semiquantitative analysis of TGF-β2 levels in control endometrium from the proliferative (n = 8) and secretory (n = 13) phases and eutopic endometrium (n = 4 per phase) and adenomyotic lesions (n = 15 per phase) from the proliferative and secretory phases with adenomyosis analyzed by immunohistochemical H-score. The results represent the mean ± SEM. ***p < 0.001. b Representative photomicrographs of immunohistochemical staining of TGF-β2 in women endometrium with (eutopic and adenomyostic lesions) and without adenomyosis during the proliferative and secretory phases. Controls represent the endometrium from women with no history or evidence of adenomyosis. Eutopic refers to the endometrium, and adenomyosis refers to lesions from women with adenomyosis. Scale bars represent 50 μm.