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. 2020 Sep 2;52(9):1550–1563. doi: 10.1038/s12276-020-00493-8

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a Proportion of all promoters and enhancers that overlap with DMRs (i.e., pDMRs and eDMRs, respectively). b Proportion of all genes targeted by pDMRs and eDMRs. c–e Significantly enriched (q value < 0.01) KEGG pathways for all DMR target genes c for eDMR target genes d and for pDMR target genes e. Immune-related, oncogenic, and metabolic-regulation KEGG pathways; cancer-associated and other pathways are marked by different color codes.