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. 2021 Apr 28;19:86. doi: 10.1186/s12915-021-01017-0

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Prdm9-deficiency changes the quantities of spermatogenic cells in all 14 tubular stages of SHR rat. Cell counts of spermatogenic cell types in seminiferous tubules of stages I to XIV are given including the mean cell number ± standard deviation per tubule in each stage calculated from 3 to 5 tubules for each stage and animal (4 mutants and 3 controls) from sections stained with PAS-H. SG A, type A spermatogonia; SG In, intermediated spermatogonia; SG B, type B spermatogonia; see legend of Fig. 4 for abbreviations of primary spermatocytes and spermatids; MII, metaphase II spermatocytes; sSC, secondary spermatocytes; dM, degenerated MI/II and sSC; dP, degenerated pachytene; dSP, degenerated, halo, and joint spermatids