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. 2021 Mar 18;46(6):1359–1371. doi: 10.1007/s11064-021-03270-9

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — UCP2 deficiency enhanced HT22 cell damage under OGD/R and high glucose conditions. a Light microscopy images showing morphological changes in HT22 cells treated with different concentrations of genipin (25 μM, 50 μM, 75 μM, and 100 μM). b Cell viability in HT22 cells at different concentrations was determined by CCK-8 assays. c, d The expression of UCP2 in the different groups at the indicated times was measured by western blotting. *p < 0.05 vs. the Control group. e Light microscopic images showing morphological changes in HT22 cells after different treatments. Scale bar = 20 μm. f Cell viability analysis of HT22 cells after different treatments. The experiments were repeated three times in triplicate for each condition. ^#p < 0.05 vs. Control; *p < 0.05 vs. HG; ^$p < 0.05 vs. NG + OGD/R; ^@p < 0.05 vs. HG + OGD/R; ^%p < 0.05 vs. NG + OGD/R + GE