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. 2021 Apr 30;12(5):428. doi: 10.1038/s41419-021-03628-5

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — A ADAMTS9 expression levels were examined by reverse transcriptase PCR assay; B and western blotting assay. C Expression levels of ADAMTS9 in AGS and BGC-823 cells transfected with ADAMTS9 overexpression plasmid and empty plasmid were analyzed by reverse transcriptase PCR and western blotting assay. D ADAMTS9 inhibits the cell growth in AGS and BGC-823 cells. E ADAMTS9 suppresses the colony formation in AGS and BGC-823 cells. F ADAMTS9 inhibited growth of tumors in vivo. Tumor growth curves and tumor weight shows the suppressive effect of ADAMTS9 in vivo. G ADAMTS9 impairs motile capacity in AGS and BGC-823 cells detected by wound healing assay (magnification 40×); H and by transwell assay (magnification 100×).