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. 2021 Apr 30;12(5):433. doi: 10.1038/s41419-021-03719-3

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — A Immunohistochemical staining analysis of POU2F2 in normal brain tissues and 12 paired GBM tissue samples. B Immunohistochemistry analyses of POU2F2 expression levels in eight normal brain samples and 12 GBM samples. C Kaplan–Meier analysis of overall survival from the R2 database with the log rank test P value indicated. D Quantitative RT-PCR analysis of POU2F2 mRNA levels in 13 paired GBM and peritumoral normal tissues. E, F Quantitative RT-PCR and western blot analysis of POU2F2 expression in normal astrocytes (SVGP12) and GBM cell lines. All data were shown as the mean ± SD, *p < 0.05, **p < 0.01. All p values were based on analysis control versus treatment.