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. 2021 May 1;12:260. doi: 10.1186/s13287-021-02353-2

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — hDPSCs attenuated damage to the articular cartilage in vivo. The macroscopic characteristics of OA model group and the hDPSC treatment group are showed in a. In addition, the results of the HE, toluidine blue staining, safranin-O/Fast Green staining, and immunochemistry of Col-II showed that hDPSCs significantly improved the histological findings of osteochondral tissue in OA models. More intense staining in the cartilage layer was showed by using arrows, respectively (a). Moreover, the high-hDPSC group exhibited significantly lower pathological scores than the other 2 experimental groups (b). **P<0.01, ***P<0.001. Bars represent 3mm in microscopic data of a and 2mm in histopathological data in a, respectively. hDPSCs, human dental pulp stem cells; Col-II, collagen II; OA, osteoarthritis