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. 2021 Apr 19;12:641848. doi: 10.3389/fphar.2021.641848

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Copyright © 2021 Song, Feng, Zhang, Deng, Yu, Zhang and Li.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Tanshinone IIA regulated NF-κB signaling pathway in BV2 cells after oxygen-glucose deprivation (OGD). (A) The protein level of p-IκB in BV2 cells treated with Tanshinone IIA. (B) The protein level of p-p65 in BV2 cells treated with Tanshinone IIA. (C) The protein level of p-IκB in BV2 cells treated with BAY-11-7082. (D) The protein level of p-p65 in BV2 cells treated with Tanshinone IIA and BAY-11–7082. ##p < 0.01, compared with the control group. *p < 0.05, **p < 0.01, compared with the OGD group. Con, control group; OGD, oxygen-glucose deprivation; TanIIA, Tanshinone IIA; BAY, BAY-11-7082, a NF-κB inhibitor, n = 3.