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. 2021 Apr 29;14:2879–2892. doi: 10.2147/OTT.S299234

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© 2021 Chen et al.

This work is published and licensed by Dove Medical Press Limited. The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed. For permission for commercial use of this work, please see paragraphs 4.2 and 5 of our Terms (https://www.dovepress.com/terms.php).

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BECN1 interfered with migration, invasion and EMT regulated by miR-140-3p. (A) A wound healing assay was used to detect the migration of BGC-823 and MKN45 cells; scale bar = 100 μm. (B) A Transwell assay was used to detect cell invasion; scale bar = 100 μm. (C) The protein expression of Vimentin, N-cadherin, E-cadherin, and LC3 II/I was determined using Western blot analysis. *P<0.05; **P<0.01; ***P<0.001.

Abbreviations: si-BCL2, siRNA targeting BCL2; pc-BECN1, BECN1-expressing vectors; NC, negative control.