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. 2021 Apr 22;14(4):100535. doi: 10.1016/j.waojou.2021.100535

Table 3.

rhC1-INH dosing.

All patients (N = 71) All attacks (N = 2356) First attack (n = 71) Single location (n = 2242) Two locations (n = 109) Three locations (n = 5)
Dose, mean
 Number of units (U) 3307 3107 3336 2749 2520
 U/kg 43.3 40.7 43.6 38.0 35.1
Number of doses, n (%)
 One 2351 (99.7) 70 (98.6) 2239 (99.9) 107 (98.2) 5 (100)
 Two
5 (0.2)
1 (1.4)
3 (0.1)
2 (1.8)
0 (0)
Patients <84 kg (n = 49)
All attacks (n = 1842)
First attack (n = 49)
Single location (n = 1741)
Two locations (n = 96)
Three locations (n = 5)
Dose, mean
 Number of units (U) 3083 2873 3113 2575 2520
 U/kg 43.5 42.0 43.9 37.3 35.1
Number of doses, n (%)
 One 1838 (99.8) 48 (98.0) 1738 (99.8) 95 (99.0) 5 (100)
 Two
4 (0.2)
1 (2.0)
3 (0.2)
1 (1.0)
0 (0)
Patients ≥84 kg (n = 22)
All attacks (n = 514)
First attack (n = 22)
Single location (n = 501)
Two locations (n = 13)
Three locations (n = 0)
Dose, mean
 Number of units (U) 4110 3627 4112 4038
 U/kg 42.7 38.6 43.0 42.5
Number of doses, n (%)
 One 513 (99.8) 22 (100) 501 (100) 12 (92.3)
 Two 1 (0.2) 0 (0) 0 (0) 1 (7.7)

During 1 HAE attack, rhC1-INH dosing was limited to 14 U/kg (total dose, 1050 U) because the patient presented with poor venous access; this oro-facial-pharyngeal attack showed improvement within 4 h. Also, during 1 HAE attack, a patient received 3 consecutive vials, recorded as a single treatment, for a total rhC1-INH dose of 117 U/kg. HAE: hereditary angioedema, rhC1-INH: recombinant human C1-esterase inhibitor