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. 2021 Feb 16;15(5):1391–1411. doi: 10.1002/1878-0261.12917

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© 2021 The Authors. Molecular Oncology published by John Wiley & Sons Ltd on behalf of Federation of European Biochemical Societies.

This is an open access article under the terms of the http://creativecommons.org/licenses/by/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Fig. 7 — Upregulation of CPT1A in CAFs is responsible for intraperitoneal tumor dissemination and growth. (A) Experimental schema used to treat HCT116‐luc + cells before i.p. injection into BALB/C nude mice. (B) Animals from each test group were bioimaged (Xenogen IVIS system) by detecting the luciferase emission spectrum at 1 week post i.p. injection to visualize the progression of tumor growth in the peritoneum. (C) Animals from each test group were bioimaged (Xenogen IVIS system) by detecting the luciferase emission spectrum at 2 weeks post i.p. injection to visualize the progression of tumor growth in the peritoneum. (D) Summary of metabolic changes that occur in interacting colon cancer cells and CAFs as described in the text.