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. 2021 May 5;21:249. doi: 10.1186/s12935-021-01953-5

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Expression profile of RAD51 in ovarian cancer. a Clinicopathological characteristics of TCGA ovarian cancer patients and genetic signatures of RAD51 were studied in detail and depicted as oncoprints using cBioPortal. b Comparison of RAD51 expression between TCGA ovarian cancer tissues and matched GTEx normal tissues in GEPIA (one-way ANOVA). c Proportion of patients who developed new neoplasm events post initial therapy was analyzed in cBioPortal. d The effect of RAD51 expression on disease-free months of ovarian cancer patients was evaluated and survival analysis was performed (log-rank test). P value was denoted as *P < 0.05