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. 2021 Feb 15;32(4):331–347. doi: 10.1091/mbc.E20-07-0492

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© 2021 Lee et al. “ASCB®,” “The American Society for Cell Biology®,” and “Molecular Biology of the Cell®” are registered trademarks of The American Society for Cell Biology.

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FIGURE 7: — Balancing and synthetic interactions between mec-7 neo and anti alleles. (A) TRN morphology of animals carrying C303Y R391H double mutations in mec-7. Arrowhead points to the absence of long ALM-PN. (B) R391H single mutation caused an anti phenotype with severely shortened TRN neurites. Arrows point to the premature termination of ALM-AN and PLM-AN. The lower panels show the excessive short sprouts growing out of PLM-AN or PLM cell body in mec-7(R391H) mutants. (C) Schematic diagram of ALM and PLM morphologies in mec-7 neo and anti single mutants, as well as their double mutants. (D) The positions of R391 (red) and C303 (yellow) mutated in the two alleles. (E) Overextension of PLM-PN and excessive neurite sprouting from PLM cell body in the mec-7(C303Y R391H) double mutants. The diagrams on the right illustrate the abnormal PLM morphology at the tail.