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. Author manuscript; available in PMC: 2022 May 1.
Published in final edited form as: Exp Hematol. 2021 Feb 22;97:66–78.e6. doi: 10.1016/j.exphem.2021.02.010

Figure 4. P53 stabilization, cell cycle inhibition, and DNA damage checkpoint pathways are upregulated in Rpl11 haploinsufficient erythroid precursor cells independent of heme content.

Figure 4.

GSEA enrichment levels for individual cells (BFU-E through basophilic erythroblasts) grouped according to clusters A-D from wildtype control mice, Flvcr1-deleted mice, and Rpl11 haploinsufficient mice shown as FDR plots for each pathway.