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. 2021 May 7;23(6):57. doi: 10.1007/s11886-021-01489-0

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Representative case of left-dominant arrhythmogenic cardiomyopathy variant in a patient with a desmoplakin-gene mutation. The left-dominant arrhythmogenic cardiomyopathy variant may show an unremarkable electrocardiogram except for low-QRS voltages in the limb leads (a) and normal dimension and systolic function of both ventricles (end-diastolic frame of cine cardiac magnetic resonance sequence in long-axis 4-chamber view). b Post-contrast cardiac magnetic resonance images disclose myocardial fibrosis in the form of a late gadolinium enhancement stria in the epicardium of the left ventricular lateral wall (arrowheads) and midmural layer of the interventricular septum (arrows) (c) (reproduced with permission from: De Lazzari M et al. J Am Heart Assoc. 2018 Nov 20;7(22):e009855. doi: 10.1161/JAHA.118.009855) [27]