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. 2021 May 7;23(6):57. doi: 10.1007/s11886-021-01489-0

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — Role of intense sports activity in the natural history of arrhythmogenic cardiomyopathy, from desmosomal-gene mutation to phenotypic expression and cardiac arrest due to ventricular fibrillation. Sports activity may promote development of phenotypic expression, accelerate disease progression, and trigger life-threatening ventricular arrhythmias (reproduced with permission from: Corrado D, Zorzi A. Eur Heart J 2015 Jul 14;36(27):1708-10. doi: 10.1093/eurheartj/ehv183. Epub 2015 May 12, with permission from Oxford University Press) [32]