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. 2021 Apr;9(8):684. doi: 10.21037/atm-21-1070

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2021 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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Verification of the targeting relationship between PPARγ and SFRP5. (A,B) Luciferase activity of each group; (C) the targeting relationship between PPARγ and SFRP5 was verified by CHIP; (D) fold enrichment of SFRP5. *P<0.05 vs. pGL-3-Basic group; aP<0.05 vs. pGL-3-1000 group; ^#P<0.05 vs. DMSO group; bP<0.05 vs. GW9662 group; cP<0.05 vs. RSG + AD-PPARγ group; ^ΔP<0.05 vs. IgG group. PPARγ, peroxisome proliferators-activated receptors γ; DMSO, dimethyl sulfoxide; GW9662, PPARγ antagonist; RSG, rosiglitazone; AD, adenovirus.