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. 2021 May 6;9:e10928. doi: 10.7717/peerj.10928

Table 2. Clinical characteristics of the LOPD patients.

During the study period patient 1-9 received alglucosidase alfa.

Patient Sex Age Year of diagnosis Start on ERT Aids Gene mutation
1 M 62 2000 2007 B-pap at night GAA-mutation: c*-32-13T>A GAA-mutation: c.1003G>A
2 F 43 2010 2011 ÷ GAA-mutation: c-32-13T>A GAA-mutation: c.307T>G; pC103G
3 F 40 2015 2015 ÷ GAA-mutation: c.32-13T>G, Homozygous
4 M 42 2015 2015 ÷ GAA-mutation: c.32-13T>G GAA-mutation: c.525 del, p. Glu176Argfs*45
5 M 44 2010 2011 ÷ GAA-mutation: c.32-13T>A GAA-mutation: c.2331+2T>G
6 F 30 2016 2016 Wheel chair for longer distance GAA-mutation: c-32-13T>G GAA-mutation: c-525 del, p. Glu176Argfs*45
7 F 25 2013 2014 Wheel chair for longer distance GAA-mutation: c-32-13T>G GAA-mutation: c-525 del, p. Glu176Argfs*45
8 M 19 2014 2015 ÷ GAA-mutation: c.32-13T>G GAA-mutation: c.1548G>A
9 F 34 2003 2014 ÷ GAA-mutation: c.32-13T>G GAA-mutation: c.1802C>T
10 M 22 1995 2006 ÷ GAA-mutation: IVS1-13T >G GAA-mutation: 2.228A>G

Notes.

During the study period patient 1–9 received alglucosidase alfa. Patient 10 received avalglucosidase alfa (neoGAA).

ERT
Enzyme replacement therapy