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. 2021 May 9;226(3):152093. doi: 10.1016/j.imbio.2021.152093

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© 2021 The Authors

Since January 2020 Elsevier has created a COVID-19 resource centre with free information in English and Mandarin on the novel coronavirus COVID-19. The COVID-19 resource centre is hosted on Elsevier Connect, the company's public news and information website. Elsevier hereby grants permission to make all its COVID-19-related research that is available on the COVID-19 resource centre - including this research content - immediately available in PubMed Central and other publicly funded repositories, such as the WHO COVID database with rights for unrestricted research re-use and analyses in any form or by any means with acknowledgement of the original source. These permissions are granted for free by Elsevier for as long as the COVID-19 resource centre remains active.

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Fig. 4 — Comparison of the decrease of CR1/E density at first determination among COVID-19 patients according to CR1 density genotypes. (A) Among CR1/E (low) from patients at the lowest value during longitudinal follow-up (Mean = 372 SD = 134 N = 52). HH: patients homozygous for the high density genotype (Mean = 426 SD = 122 N = 32). LH patients heterozygous for the high and low density genotypes (Mean = 308 SD = 93 N = 17). (B) CR1/E%: CR1/E were expressed as the percentage of the mean CR1/E density predicted from the genotype of a given individual. Among CR1/E (low) from patients at the lowest value during longitudinal follow-up (Mean = 60 SD = 18.3 N = 52). HH% (Mean = 57 SD = 16.3 N = 32); LH% (Mean = 66 SD = 19.9 N = 17). Means are depicted as open squares. Proportionally, patients with the HH high density genotype displayed a more important loss of CR1/E than LH heterozygous patients. Wilcoxon median test (p < 0.001).