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. 2021 Apr;13(4):2447–2459. doi: 10.21037/jtd-21-212

Figure 6.

Figure 6

DAPK1-plasmid reversed the roles of miR-141 in hypoxia-induced cardiomyocyte injury. MiR-141 mimic, mimic control, control-plasmid, or DAPK1-plasmid were transfected into hypoxia-induced H9c2 cells for 48 h. The secretion of (A) CK-MB and (B) cTnI in hypoxia-induced cardiomyocytes was determined. (C) Mitochondrial activity was evaluated in different groups. Results were presented as mean ± SD. **, P<0.01 vs. hypoxia + mimic control; ##, P<0.01 vs. hypoxia + miR-141 mimic + control-plasmid.