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. Author manuscript; available in PMC: 2022 Jul 1.
Published in final edited form as: J Comp Neurol. 2021 Feb 15;529(10):2750–2770. doi: 10.1002/cne.25121

Figure 12.

Figure 12.

Myelin and oligodendrocyte markers that did not correlate with diffusion tensor imaging in striatum of piglets with subacute injury. (a) The number of large CNPase+ myelin swellings in the internal capsule did not correlate with mean diffusivity (MD; r=0.000, p>0.999). (b) The number of degenerating BIN1+ cells (r= −0.662, p=0.061) and (c) the ratio of degenerating BIN1+ cells to total BIN1+ cells (r= −0.662, p=0.061) in internal capsule were not associated with fractional anisotropy (FA). In caudate, the ratios of (d) normal-to-total CNPase cells (r= −0.433, p=0.250), (e) doublet-to-total CNPase cells (r=0.469, p=0.204), (f) apoptotic-to-total CNPase+ cells (r=0.075, p=0.853), and (g) apoptotic-to-total BIN1+ cells (r=0.034, p=0.946) did not correlate with MD. Symbols indicate piglets that received QA (open circle: 240 nmol, solid circle: 720 nmol, and solid triangle: 960 nmol), HI (solid squares), or sham procedure (open square).