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. 2021 Apr 28;8:655157. doi: 10.3389/fmolb.2021.655157

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Copyright © 2021 Liu, Li, Bai and Xu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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XIST induces autophagy and inhibits oxidative stress in the HG-treated Schwann cells. (A–D) The RSC96 cells are treated with HG, or co-treated with HG and pcDNA-3.1-XIST overexpression vectors. (A) The expression of XIST is measured by qPCR assays. (B,C) The expression of LC3I, LC3II, and Beclin-1 is assessed by Western blot analysis. (D) DCFH–DA staining shows the ROS production. N = 3, mean ± SD, *P < 0.05, **P < 0.01.