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. 2021 Apr 28;12:609693. doi: 10.3389/fphar.2021.609693

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Copyright © 2021 Zhao, Li, Gao, Xu, Zhu, Zhai, Zhang, Shen, Di, Wang, Chen, Huang, Sun and Liu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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BBR inhibits CNPY2-mediated ERS pathway. The expression of CNPY2 and GRP78 in ischemic brain tissue was analyzed by immunofluorescence staining to further evaluate the effect of BBR on the ERS pathway. (A), The location of CNPY2 in the ischemic penumbra of each group. (B), Ratio of fluorescence intensity of CNPY2 in ischemic penumbra. (C), The location of CNPY2 in the ischemic penumbra of each group. (D), Ratio of fluorescence intensity of CNPY2 in ischemic penumbra. Original magnification is 400×, scale bars is 20 μm n = 6. Data are shown as the mean ± SEM. *p < 0.05 compared with the Sham group; ^# p < 0.05 compared with the CIRI group.