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. 2020 Nov 18;3(1):acmi000179. doi: 10.1099/acmi.0.000179

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© 2021 The Authors

This is an open-access article distributed under the terms of the Creative Commons Attribution NonCommercial License. This article was made open access via a Publish and Read agreement between the Microbiology Society and the corresponding author’s institution.

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Fig. 1. — The overlap of preterm infant AMR contigs across four ST131 subclade C2 assemblies (8289_1#3, 8289_1#24, 8289_1#27, 8289_1#34), two ST131 reference genomes (EC958 and SE15) and two HMP assemblies (83 972 and 3_2_53FAA). Top: The intersection sizes (y-axis) and the numbers of AMR contigs per set showed that most (210) AMR contigs were shared across all isolates. The non-unique AMR contigs indicated smaller numbers of unique AMR genes, so EC958’s 17 AMR contigs (red) corresponded to nine unique bla _CMY genes (Table S2); and 8289_1#24’s nine contigs (green) represented four aminoglycoside and tetracycline resistance genes (Table S3). All Clade C bar 8289_1#27 had 12 contigs (blue) denoting to five bla _TEM genes (Table S4). Bottom: The numbers of AMR contigs per sample with the corresponding sets (coloured circles).