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. 2020 Nov 17;121(5):573–583. doi: 10.1055/s-0040-1720980

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The Author(s). This is an open access article published by Thieme under the terms of the Creative Commons Attribution-NonDerivative-NonCommercial License, permitting copying and reproduction so long as the original work is given appropriate credit. Contents may not be used for commercial purposes, or adapted, remixed, transformed or built upon. ( https://creativecommons.org/licenses/by-nc-nd/4.0/ )

This is an open-access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivatives License, which permits unrestricted reproduction and distribution, for non-commercial purposes only; and use and reproduction, but not distribution, of adapted material for non-commercial purposes only, provided the original work is properly cited.

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Fig. 2 — Schematic illustrations of F7 and F10 depicting DNA methylation patterns and location of correlated CpGs in blood and liver tissue. At least 5 kbp upstream and 0.5 kbp downstream of each gene are included: blue indicates the gene structure and red CpG islands. Variable CpGs and those displaying moderate and strong correlations are shown in gray . The average methylation status of the 27 samples for each CpG in blood and liver is also presented; purple represents a low degree of methylation (hypomethylation) and green a high degree of methylation (hypermethylation). All other genes can be found in Supplementary Figure S2 (available in the online version).