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. 2021 Apr 21;40(19):3422–3433. doi: 10.1038/s41388-021-01656-1

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — A Paired normal stomach (NS), primary tumors (PT), and peritoneal metastases (PM) were collected from 6 gastric cancer patients and submitted for transcriptome sequencing. B PCA and (C) unsupervised, hierarchical clustering of normalized counts from transcriptome sequencing of NS, PT, and PM. D Four candidate genes (S100A13, BCHE, ITGA4, and LIMK1) were selected from top up-regulated genes in peritoneal metastases compared to primary gastric cancer. All four genes have been reported having oncogenic functions and have inhibitor(s) available.