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. 2020 Nov 20;24(3):391–401. doi: 10.1007/s00737-020-01093-1

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — External environmental cues can stimulate neuronal calcium (Ca²⁺) influx via effects on extracellular matrix components (notably CCN proteins and hemicentin (HMCN1)), a variety of transmembrane receptors, and intracellular signalling cascades; the expression/activity of many of these proteins is differentially sensitive to sulfated and free steroids, and/or is affected by steroid sulfatase (STS) deficiency (indicated with an asterisk symbol)