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. 2021 May 13;17(5):e1009516. doi: 10.1371/journal.pgen.1009516

Fig 5. Retention of RNA editing at intronic sites in Adar1 p110–specific KO and Adar1 p110/Adar2 double KO mice.

Fig 5

(A, B) The mean values for the retention of RNA editing at intronic sites in brains from two Adar1 p110-specific KO (Adar1 p110-/-) (A) and two Adar1 p110/Adar2 double KO (Adar1 p110-/- Adar2-/-) (B) mice at post-natal day 0 (P0). Retention was calculated only for sites where the mean editing ratios of two wild-type (Adar1 p110+/+) mice were more than 5%, which are displayed on the vertical axis. (C, D) The mean values for the retention of RNA editing at intronic sites in thymi from two Adar1 p110-/- (C) and two Adar1 p110-/- Adar2-/- (D) mice at P0. Retention was calculated only for the sites where the mean editing ratios of two Adar1 p110+/+ mice were more than 5%, which are displayed on the vertical axis. (E, F) The intronic region encompassing RNA editing sites (indicated by a red box) of Fnbp1 (E) and Dlg4 (F) genes was analyzed using the University of California Santa Cruz (UCSC) genome browser.