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. 2021 May 14;166(7):2005–2010. doi: 10.1007/s00705-021-05096-w

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — Anti-HEV activity of ivermectin in different human cell culture models. The effects of ivermectin treatment on viral-replication-dependent luciferase activity, the 50% inhibitory concentration (IC₅₀) against HEV replication, and the 50% cytotoxic concentration (CC₅₀) of ivermectin in different cells were calculated using GraphPad Prism 5 software. (A) 293T kidney cells (n = 6-12) (B) PLC/PRF/5 cells (n = 10-12) (C) U87 neuronal cells (n = 10-12). The untreated (CTR) group served as control (set as 100%). RLU: relative luciferase units. Data are presented as the mean ± SEM (*, P < 0.05; **, P < 0.01; ***, P < 0.001).