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. 2021 May 14;12(5):489. doi: 10.1038/s41419-021-03788-4

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — A Less circulatory and hepatic monocytic MDSCs in CXCL10^−/− mice compared to wild-type ones. B Significantly decreased mobilization and recruitment of monocytic MDSCs in TLR4^−/− mice. C No significant change of circulatory and hepatic monocytic MDSCs by knockout of CXCR3. D Reduced TLR4⁺ monocytic MDSCs in the circulation and liver of CXCL10^−/− mice compared to wild-type ones after IRH. N = 5/group; Error bars indicate Standard Error of Mean; *p < 0.05, **p < 0.01. MDSC myeloid-derived suppressor cell, IRH ischemia/reperfusion plus major hepatectomy, WT wild type.