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Effects of E2 and TGFβ on ERα activation and TGFβRII expression of BMET-derived ER+ breast cancer cells. (A) Constitutive and E2-stimulated (S104/106 and S118 phosphorylated) ERα and (B) TGFβRII expression in 43-4M cells maintained in E2-deplete media for 4 days prior to simultaneous treatment with E2 (10−8 M) and/or TGFβ (5 ng/mL) for the indicated times.
