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. 2021 May 13;19(2):15593258211011023. doi: 10.1177/15593258211011023

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© The Author(s) 2021

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 6. — TEH inhibited the progression of HCC through PI3K/Akt/mTOR signaling pathway. Huh-7 and HepG2 cells were treated with TEH (5 μM) and/or PI-103 (1 μM) and then subjected to irradiation (4 Gy). (A-B) CCK8 experiment was carried out to examine the proliferation of Huh-7 and HepG2 cells. (C) The apoptosis rate of Huh-7 and HepG2 cells was evaluated via flow cytometry. (D-E) Western bolt was applied to measure autophagy-related proteins and PI3 K / Akt / mTOR pathway expression. *P < 0.05, **P < 0.01, ***P < 0.001 (vs.4 Gy + DMSO group); ns P > 0.05 (vs.4 Gy + PI-103 group).