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. 2021 May 18;11:10548. doi: 10.1038/s41598-021-89981-z

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Suppression of miR-378c in human atherosclerosis. (A) Heatmap showing MicroRNAs expression profiles in human normal coronary artery and artery with plaques. The colors indicate the ln-transformed FPKM values. (B) MicroRNA array analysis for human normal coronary artery and artery with plaques. Each point represents FPKM value for individual MicroRNA. MicroRNAs that were significantly up-regulated and down-regulated in artery with plaques were highlighted in red and blue, respectively. (C) Heatmap showing top differentially regulated MicroRNAs (P < 0.05) in human artery with plaques relative to normal controls. (D) qRT-PCR analysis of miR-378c expression in atherosclerotic plaques tissues (n = 6) and normal controls (n = 5). Data were presented as mean ± SD. (E) Blood miR-378c levels were measured in 215 ACS patients and 52 healthy subjects. Data were presented as mean ± SD. *P < 0.001 as compared between two groups.