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. 2021 May 18;14:26. doi: 10.1186/s13039-021-00544-3

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — STR genotype results. A set of 21 polymorphic STR markers, including D3S1358, TH01, D21S11, D18S51, Penta E, D5S818, D13S317, D7S820, D16S539, CSF1PO, Penta D, vWA, D8S1179, TPOX, FGA, D19S433, D12S391, D6S1043, D2S1338, D1S1656 and Amelogenin, was used. STR genotype analysis using DNA from cells suspended in Carnoy's fixative fluid after karyotyping (a) and DNA from the CMA test (b) verified the concordance between the two samples in case 2 (five markers are shown in this image) and demonstrated that the samples indeed were from the same individual