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. 2021 May 19;7:117. doi: 10.1038/s41420-021-00498-w

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — A The effect of SRA1 on CD44 Transcription. B The effect of SRA1 on CDH1 Transcription. C The effect of SRA1 on TP53 Transcription. D The effect of SRA1 on CAV1 Transcription. E, F The effect of overexpressed SRA1-L/S on downstream CD44, AKT, and ERK signaling pathway proteins was detected in HepG2 cells by western-blot. G, H The effect of knocked down SRA1-E3, SRA1-E2E4, and SRA1-In1 on downstream CD44, AKT, and ERK signaling pathway proteins was detected in HepG2 cells by western-blot. Data are presented as mean ± S.D. (N = 3). The “*, **, ***” indicate “P < 0.05, 0.01, 0.001” versus the control group, respectively. “^#, ^##, ^###” represents “P < 0.05, 0.01, 0.001”.