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. 2021 May 21;19:150. doi: 10.1186/s12951-021-00894-5

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — PGZ-exos protected against HG-induced inhibition of HUVECs viability and cell proliferation. A CCK8 assay was performed to assess HUVECs viability after treatment with HG and exosomes. B, C EdU incorporation assay was used to evaluate HUVECs proliferation. Scale bar: 50 m. D, E Flow cytometry was used to assess the cell cycle distribution of the HUVECs. F, G Western blotting was used to investigate the protein expression of Cyclin D1, Cyclin D3 and VEGF. Data are presented as meansSD from three independent experiments. *P<0.05 versus the control (con) group