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. 2021 Jan 6;44(3):956–964. doi: 10.1007/s10753-020-01390-y

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — Non-pregnant and pregnant mouse brain IMCs and microglial activity at basal state, following inflammation with or without progesterone pretreatment. a microglia was gated as CD45^low; brain hematopoietic cells were gated as CD45^high. b–d CD45^highCD11b⁺Gr1⁺ IMCs in non-pregnant mice: Control, LPS, LPS + P respectively. e–g CD45^highCD11b⁺Gr1⁺ IMCs in pregnant mice: Control, LPS, LPS + P respectively. h the percentage of CD11b⁺Gr1⁺ IMCs out of CD45^high hematopoietic cells are plotted as mean ± SD. i microglial activity measured as medial CD45 intensity in CD45^low cells plotted as mean ± SD. *p < 0.05, compared to the non-pregnant LPS group; **p < 0.05, compared to the pregnant LPS group.