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. 2021 May 22;12(6):529. doi: 10.1038/s41419-021-03796-4

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© The Author(s) 2021, corrected publication 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — A MTT assay was employed to show the effects of DPP3 on cell proliferation of RKO and HCT 116 cells. B, C Flow cytometry was performed to detect cell cycle (B) and cell apoptosis (C) of RKO and HCT 116 cells with or without DPP3 knockdown. D Human apoptosis antibody array was utilized to analyze the regulatory ability of DPP3 on expression of apoptosis-related proteins in HCT 116 cells. E The protein expression of downstream molecular in HCT 116 cells with or without DPP3 knockdown was measured by WB. F, G CRC cell migration ability was accessed by Transwell assay (F) and wound-healing assay (G). The representative images were selected from at least three independent experiments. Data were shown as mean ± SD. *P < 0.05, **P < 0.01, ***P < 0.001.