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. 2021 May 4;10:e64213. doi: 10.7554/eLife.64213

Figure 4. Maps of fibrotic tissue distribution (left) and activation time (right) for ESUS and AFib models in which pacing succeeded (rows 1–2) or failed (row 3) to induce RD-driven arrhythmia.

Figure 4.

Black arrows indicate directions of wavefront propagation in RDs. Double lines indicate sites of conduction block. Black-shaded regions in activation maps indicate locations where activation did not occur during the analysis interval. (A) ESUS model with 6.9% fibrosis and reentry inferior to LIPV. (B) ESUS model with 10.0% fibrosis and reentry on the atrial floor. (C) ESUS model with 16% fibrosis with wavefront termination through fibrosis on posterior wall. (D) AFib model with 9.9% fibrosis and reentry observed adjacent to RIPV on posterior wall. (E) AFib model with 13.7% fibrosis and reentry observed on the anterior wall. (F) AFib model with 11.6% fibrosis with wavefront collision on posterior wall.