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. 2020 Dec 15;26(4):881–890. doi: 10.1007/s10741-020-10063-9

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© The Author(s) 2021, corrected publication 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Central illustration. Oxidative stress and inflammation in anthracycline cardiotoxicity.¹⁸F-FDG PET, ¹⁸F-fluoro-D-glucose positron emission tomography, CMR cardiovascular magnetic resonance, DPP4i dipeptidyl-peptidase 4 inhibitor, hs-CRP high-sensitivity C-reactive protein, IgE immunoglobulin E, IL interleukin, MPO myeloperoxidase, ROS reactive oxygen species, SPECT single-photon emission computed tomography, TAOS total antioxidant status, TNFα tumor necrosis factor alpha, TTE transthoracic echocardiogram