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. 2021 May 26;21:152. doi: 10.1186/s12906-021-03322-1

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Chondrocytes were stimulated with IL-1β to construct OA model in vitro. a, b Cell viability was measured by CCK-8 method after CHON-001 and ATDC5 cells were treated with 0, 1, 5, 10 ng / mL IL-1β for 24 h. c CHON-001 and ATDC5 cell lines were treated with 0, 1, 5, 10 ng / mL IL-1β, and the apoptosis of CHON-001 and ATDC5 cells was detected by flow cytometry. d-f CHON-001 and ATDC5 cell lines were treated with 0,1,5 and 10 ng/mL IL-1β, and the contents of IL-6, IL-8 and TNF-α were detected by ELISA. *P < 0.05, **P < 0.01, ***P < 0.001 vs. control group.