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. 2021 May 27;11:11127. doi: 10.1038/s41598-021-90508-9

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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DECT images of a 52-year-old man with HCC undergoing lipiodol TACE. The right hepatic lobe showed the original lesions were completely deposited with lipiodol, and the new lesions appeared, a low-attenuation nodule on axial unenhanced CT image (A), significant enhancement at the arterial phase (B), and wash-out at the portal venous phase(C). ROIs of TAA and ANHP (D and E) placed on arterial and portal iodine map showed IC and NIC of TAA to be 2.3 mg/ml and 19.2% at arterial phase, and 2.1 mg/ml and 42.5% at portal venous phase. IC and NIC of ANHP was 0.1 mg/ml and 1.0% at arterial phase, and 2.1 mg/ml and 42.9% at portal venous phase. The λ_Hu of TAA and ANHP was 3.24 and 0.86, respectively (F).