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. 2021 May 28;22:200. doi: 10.1186/s12882-021-02400-3

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Partial effects plots of the difference in hazard ratio between AKI and no AKI across levels of BMI with a CKD incidence b CKD progression and ESRD c death and d combined CKD outcome. The plot is based on the multivariable Cox model that includes terms for AKI status, continuous BMI as a restricted cubic spline, study center, baseline CKD status, ICU status, diabetes, age, cardiovascular disease, race, gender, smoking, sepsis, chronic heart failure and the interaction between AKI status and BMI as a restricted cubic spline. Covariate values for the reference patient include age (65 years), race (white), gender (male), diabetes (no), hypertension (yes), smoker (former), CHF (no), CVD (no), ICU (no), and sepsis (no)